It has been reported that cancer has a lot more potential cures than previously believed! Check out the latest data about the amazing effects of a miracle drug!
Here is what a post on social media platform X notes featuring a video of Joe Rogan:
“The Overlooked Miracle Drug for Cancer? Why Big Pharma Fears Fenbendazole
“Fenbendazole has at least 12 proven anti-cancer mechanisms in vitro and in vivo,” wrote cancer researcher Dr.
@MakisMD
.”So why are there no Fenbendazole clinical trials for cancer? The answer seems rather obvious: it’s very cheap, it’s safe, and it seems to be very effective. Fenbendazole is not going to make anyone rich, and in cancer treatments, that is a non-starter.”
“What an enemy of the people,”
@JoeRogan
responded. “They’re preventing information and preventing people from using things that save them.””
The Overlooked Miracle Drug for Cancer? Why Big Pharma Fears Fenbendazole
“Fenbendazole has at least 12 proven anti-cancer mechanisms in vitro and in vivo,” wrote cancer researcher Dr. @MakisMD.
“So why are there no Fenbendazole clinical trials for cancer? The answer seems… pic.twitter.com/CAPIXoKY39
— The Vigilant Fox 🦊 (@VigilantFox) October 31, 2023
We can also look at the official data presented by NIH:
Recent studies have shown that benzimidazole anthelmintic agents can be repurposed to treat cancers that are resistant to conventional therapies. In order to evaluate the effectiveness of benzimidazole on resistant cells, a study was conducted on 5-fluorouracil-resistant colorectal cancer cells.
Different cell death pathways were investigated, and the viability of both wild-type and 5-fluorouracil-resistant SNU-C5 colorectal cancer cells was assayed.
Western blotting was performed to analyze the results. Additionally, flow cytometry assays were used to analyze both cell death and cell cycle. The study found that fenbendazole, when compared with albendazole, was more effective against 5-fluorouracil-resistant SNU-C5 cells and was thus used in subsequent experiments.
The study found that fenbendazole could be an effective treatment for 5-fluorouracil-resistant cancer cells.
The drug caused cell cycle arrest at the G2/M phase in both wild-type and resistant SNU-C5 cells.
In comparison to the wild-type cells, the resistant cells had reduced autophagy, increased ferroptosis and ferroptosis-induced apoptosis, as well as less activation of caspase-8 and p53.
The results suggest that the anticancer activity of fenbendazole does not depend on the presence of p53 in 5-fluorouracil-resistant SNU-C5 cells.
This is beyond amazing news and it should give hope to people all over the world.
